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I read that chronich wasting disease has been confirmed in Texas. I hope that it hasn't spread to Louisiana! I would send those pics to LDWF. If you kill one that looks this way you may consider submitting the brain and or spinal chord. What area was this picture taken in?
Cwd has been found i west texas about 1,000 miles from here, no evidence anywhere near the south. EHD, is common here and most deer are immune to it, very few die from it here, and certainly not in a wet year. Looking at this deer, even tho its obviously in very poor condition, it does not look 'sick', the eyes look clear and no signs of distress. Seems like i remember that huuricanes can deposit lots of flukes(worms) on vegetation. When the deer accidently consume these while browsing and they infest the stomach so the deer starves to death even with plenty to eat. While this is obviously a problem now, it will not cause long term damage to the herd.
Trail camera picture was taken not far from Lake Arthur.
Unfortunately last month we stumbled on a young dead deer while preparing our deer stands. Similar to what the article stated we also have a few hundred pictures of a sickly doe that has gorged herself on ricebrand just to get skinnier. Its a sad sight and I hope to be able to put her down opening morning of rifle season. I'm checking my cameras again on another area of our property on Friday and my fingers are crossed I don't see more deer affected.
Chronic wasteing disease has never been found in the south, and its never been passed from one deer to another and very rarely in wild deer anywhere. 99+% of deer and elk were pen raised deer. Its never been found in a young deer, the deer/elk in pens were fed high protein/mineral diets.Most of the wild cases were in extremely dry areas. This leads some to believe cwd is not a disease at all, but a naturaly occurring condition caused by long term ingestion of high levels of minerals, causeing a toxicity problem leading to starvation, or chronic wasteing. Not many deer in any area have ever tested positive.
So how exactly is it spread? Is it just a spontaneous reaction? Since it was confirmed in Texas, how fast would it take to get to La? And also what is this disease?? I am curious?? Are you a biologist? If so could you point me in the direction to some good references that may help me research this topic further? I think it would make a good paper!
No i'm not a biologist, just a serious deer manager, so i try to read everything written about deer. Great articles in texas trophy hunters magazine on this subject, read them and it will give u a good understanding. I recently visited the ares where this occurred in colo., texas, and wyoming to see for myself, and we stayed at a holiday inn,lol and i like i said i've studied this since it first showed up in wisconsin in the wild.
Guys, HUNTR may be a serious deer manager, but he has some serious misconceptions and is spouting misinformation. Several of his statements are plainly wrong. Info on CWD is easy to come by. Start here with the Center For Disease Control (CDC). http://www.cdc.gov/ncidod/dvrd/cwd/
Sorry huck, thats old news and u really need to stop believing what u read from the government and google. Read horace gore and dr. kroll in ttha mag then we'll talk. I'm sure everyone has seen what my crazy ideas have gotten me, where's ur pics?
By now i assume anyone that was concerned about this has read up on the facts. CWD never has and prolly never will be a problem in the southeast. So my question is, Why did EHD kill so many deer in southeast La. this year? Central la. was much drier much earlier than south, but few deer were reported dead. The reason deer numbers are kept so low is to prevent dieoffs from disease like this. So if they die off anyway, why keep numbers so low? An example, La. has about 30 deer per sq. mile, montana about 100. If we lose 10%, about average, it has very little effect. In montana, they can lose 50% and still have twice as many as us and theirs recover much faster. Actually the reason it kills any deer in the south is because of poor nutrition. This shows up in poor horn growth and less resistance to disease. You never see this in areas with good habitat or where they are supplemental fed.
People there has been a number of outbreaks of hemorrhagic disease this year. Parts of Louisiana were affected and some areas in the midwest were hit very hard. These pictures on all of your trail cams are not CWD.
CWD is a prion disease. It can be transmitted from deer to deer. It is not caused by high protein feeds are anything like that. If that were the case then pen raised deer all across the country would be susceptible to an outbreak.
Infected captive (cwd) herds have been destroyed their enclosures cleaned with chemicals and reintroduced deer became infected. This is because the prion if very durable and can remain in the environment for many years. It takes temperatures of about 800 degrees to destroy it.
Mike, would u explain what a prion is, no fair useing google.lol CWD has never been PROVEN to be transmitted deer to deer, and NO young deer have ever been found with it. A lot of knowledgeable people dont even believe CWD is a disease but a toxic accumulation just like mercury poisoning. Deer in the south have very good immunity to EHD'S, nutritionaly stressed deer are much more susceptable.
My understanding is a prion is a protein that has a mutation from what it is suppose to be. The causes the protein to fold in an manner that it normally wouldn't. The protein/prion destroys the brain by slowly causing a swiss cheese type effect in the brain.
Mad cow disease, scrapies (sheep) and bovine encephalopathy is a prion diseases as well and can be transmitted between cattle, sheep and people. CJD is a prion disease that occurs in people very rarely for no reason other than probably a naturally occuring mutation.
CWD takes a while for its symptoms to become visible. It varies but can be for as much as 5 years.
All of that is off the top of my head. I haven't read much on it the last couple of years.
CWD is spread via saliva and urine I believe. That is why feeders came under fire when CWD first began spreading.
It has naturally occurred in a small area out west for many many years.
Now info from duckduckgo(search engine):
Chronic Wasting Disease (CWD) is a contagious neurological disease affecting deer, elk and moose. It causes a characteristic spongy degeneration of the brains of infected animals resulting in emaciation, abnormal behavior, loss of bodily functions and death.
CWD belongs to a group of diseases known as transmissible spongiform encephalopathies (TSEs). Within this family of diseases, there are several other variants that affect domestic animals.
Several rare human diseases are also TSEs. Creutzfeldt-Jakob disease (CJD) occurs naturally in about one out of every one million people worldwide. Variant Creutzfeldt-Jakob disease (v-CJD) has been associated with the large-scale outbreak of BSE in cattle herds in Great Britain.
A prion in the Scrapie form (PrPSc) Listeni/ˈpriːɒn/ is an infectious agent composed of protein in a misfolded form. This is the central idea of the Prion Hypothesis, which remains debated. This would be in contrast to all other known infectious agents (virus/bacteria/fungus/parasite) which must contain nucleic acids (either DNA, RNA, or both).
All known prion diseases affect the structure of the brain or other neural tissue and all are currently untreatable and universally fatal.
Prions propagate by transmitting a misfolded protein state. When a prion enters a healthy organism, it induces existing, properly folded proteins to convert into the disease-associated, prion form; the prion acts as a template to guide the misfolding of more proteins into prion form. These newly formed prions can then go on to convert more proteins themselves; this triggers a chain reaction that produces large amounts of the prion form. All known prions induce the formation of an amyloid fold, in which the protein polymerises into an aggregate consisting of tightly packed beta sheets. Amyloid aggregates are fibrils, growing at their ends, and replicating when breakage causes two growing ends to become four growing ends.